NTEU Staffs' Evidence.
A condensed portion, read by Sr. V.P. to Our U.S.A. Congress' Committee Formal Hearing.
READ THESE
Yamouyiannis
Sodium Fluorosilicate
GOVERNMENT FACTS ET AL
International
Academy of Oral Medicine and Toxicology
Protocol for Mercury/Silver Filling Removal
PATIENT
PROTECTION
Protect Your Patients!
American Medical Association: Still NO Approval of Fluoridation as Safe
June 29th 2000
20-24 June 1998
The need for a Code of Ethics at United States-EPA
Co-Sponsored by the California Association of Environmental Professionals
Fluoride Damages Bones Even at Levels Found In Drinking Water
A American Dental Association "Gagging dentists" from "telling patients The Truth"
Dentist? So are these Professionals
to officials:
Letters to our Council 2001
Fluoride.
Are your children suffering?
Periodontal Disease?
Gingivitis?
Can you read between the lines? Yes? Okay.
Comment
Both the enamel and the dentin show a pair of light (hypo) and dark (hyper)
incremental layers for each injection of fluorine. Chronic fluorosis creates daily accentuations which also average 16 microns per day.
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Millions of people world-wide are afflicted.
It also affects animals foraging on contaminated feed.
- My Story, by Darlene Sherrell ... 60 years of chronic fluoride poisoning
- Excerpts from The Cause of Fibromyalgia, by Jason Uttley - February 2007
- Allergy and Hypersensitivity to Fluoride, by Bruce Spittle, FLUORIDE 26(4):267-273 October 1993
- The Pre-Skeletal Phase of Chronic Fluoride Intoxication, by George Waldbott, FLUORIDE 7(3):118-122 1974
- Trace Elements in Human and Animal Nutrition
- Review of Fluoride Benefits and Risks USDHHS 1991, Symptoms of chronic fluoride poisoning
- ADA reference: Fluorides, hydrogen fluoride, and fluorine (F) Agency for Toxic Substances and Disease Registry, USDHHS
- Excess Fluoride Ingestion And Thyroid Hormone Derangements In Children Living In Delhi, India
- Is Dental Fluorosis Caused By Thyroid Hormone Disturbances?
- Delayed Eruption of Teeth, by Philip RN Sutton, 1996
- Lifesaver's Guide to Fluoridation, by John Yiamouyiannis, PhD
- Are we brushing aside fluoride's dangers?, The Boston Globe 1999
- Central Nervous
System Damage from Fluorides
by Phyllis J. Mullenix, PhD - Aluminum Fluoride, G proteins, and the Endocrine Glands
- Aluminum-fluoride, Beryllium-fluoride, G proteins (References)
- Dental Fluorosis Photos from NAS/NRC
- World Health Organization, Fluorides and Human Health, 1970
- American Dental Association Booklet, 1939 (iodine/fluorine)
- Periodontal bone loss precipitated by fluoride (US Patent 5,807,541)
- Photos of Chronic Fluoride Poisoning in India
- Skeletal Fluorosis in India, The Manchester Guardian, July 1998
- Fluorosis makes Amreli villagers stoop
- Fluorosis · Early Warning Signs and Diagnostic Test
- Mehsana District, Gujarat State, India
- Akwesasne Mohawks
- Asthma and Fluoride
- E-mail to National Academy of Sciences, re: errors
- Repetitive Strain Injury, New Zealand Medical Journal
- Fluoride, Gingivitis and Oral Cancer
Symptoms of chronic fluoride poisoning
dental, skeletal, arthritic, gastrointestinal, etc.
Skeletal fluorosis is not easily recognisable until the disease has developed to an advanced stage. Excessive quantities of fluoride when deposited in the skeleton is more in Cancellous bone compared to cortical bone. Changes in the bone will then be revealed through radiographs. Maximum ill effects of fluoride are detected in the neck, spine, knee, pelvic and shoulder joints. It also affects small joints of the hands and feet. The usual complaints of the patients, viz., pain in the neck, back, joints and rigidity begins in regions where Cancellous bones predominates. With increased severity of skeletal fluorosis, pain is associated with rigidity and restricted movement of cervical and lumber spine, knee and pelvic joints as well as shoulder joints.
When calcium fluor-apatite deposition takes place in the bone, the bone density and bone mass get increased. In the backbone, the perforations through which nervous and blood vessels pass through, are constricted, leading to excess pressure on nerves and blood vessels culminating in paralysis and excruciating pain.
Dental fluorosis
Dental fluorosis is an aesthetic and social problem besides being a health problem. The discoloration of teeth may change the colour from white, yellow brown to black. The discoloration may be in spots, streaks invariably horizontal in orientation, as during development new layers of the matrix are added horizontally.
Enamel The outermost covering of the tooth, is the hard structure in the body, with inorganic compounds, mainly with the calcium salts. Enamel protects the tooth beside giving colour and lustre.
Normally healthy dental enamel is semi-transparent, smooth and milk white in appearance. However, appearance of white opaque patches on the enamel may be indications of initial phases of dental fluorosis. In extreme cases of fluorosis not only does the entire dental enamel turn opaque white to brown, but the teeth tend to break off easily and even their shape may begin to be affected.
Effects of fluoride/ fluorosis on soft tissues/ organs/ systems
The conventional belief that fluoride affects only bone and teeth has been negated in recent years, as the evidence on the involvement of the soft tissues/ organs/ systems of the body are convincing. Although radiographs taken on the fluorosed individuals do reveal that ligaments do calcify, very little attention was paid in past to understand the extent of soft tissue involvement in fluorosis.
Convincing evidence now demonstrate the damage or involvement of: 1) Skeletal muscle 2) Erythrocytes 3) Gastro- intestinal systems as well as 4) Ligaments in human fluorosed patients. There are evidences of involvement of other organs and systems of the animal models, viz., kidney, liver, adrenal gland and reproductive organs.
Studies have shown that skeletal muscle is directly involved in fluorosis. Muscle involvement was earlier considered as 'secondary' effect due to neuronal involvement. The electron microscopic observations and biochemical data suggests that there is primary muscle destruction in fluorosis. It is evident from patient of fluorosis that they suffer from muscle weakness, loss of muscle energy and cannot carry out normal routine work.
Effects of fluoride poisoning on the gastro-intestinal mucosa
Acute abdominal pain, diarrhoea, constipation, blood in stool, bloated feeling (gas), tenderness in stomach, feeling of nausea (flu-like symptoms) and mouth sores, loss of appetite are common complaints due to fluoride toxicity.
Fluoride is known to combine with HCl of the stomach and is converted to hydrofluoric acid. Hydrofluoric acid is highly corrosive. The stomach and intestinal lining (Mucosa) is destroyed with loss of microvilli (the structure which is responsible for absorbing the nutrients from food), drying up and cracking of the cell surface and mucus (the slimy substance required for comfortable bowl movements) production is hampered.
Neurological manifestations
Nervousness, depression, tingling sensation in fingers and toes, excessive thirst (Polydypsia) and tendency to urinate frequently (Polyurea) are controlled by certain regions of brain that appears to be adversely affected.
Allergic manifestations
Very painful skin rashes which are perivascular inflammations, pinkish red or bluish red round or oval shaped spots on the skin prevalent in women and children that fade and clear up in 7-10 days can also occur.
Urinary tract manifestations
Urine may be much less in volume yellow-red in colour and itching in the genitals may occur.
Ligaments and blood vessels calcification
A unique feature of the disease is that soft tissues like ligaments, blood vessels tends to harden and calcify and blood vessels get blocked. Calcified ligaments and blood vessels can be seen in radiographs.
SOURCE: Depleting Groundwater Levels and Increasing Fluoride Concentration in Villages of Mehsana District, Gujarat, India: Cost to Economy and Health, Project Report, May 1998
When correlated with the analysis reports, the health complaints from the survey indicated that the most common complaints viz., body pain, knee pain and back pain were prevalent among people who consumed water containing fluoride at concentrations of 1.10 ppm which is slightly higher than the desirable limit of 1 ppm. This may be due to the chronic toxicity effect of fluoride.
The signs and symptoms exhibited by the villagers indicates clearly that they are suffering from skeletal/dental fluorosis. Since there are no industries in the vicinity of these villages, the flouride in the water is probably from the earth’s crust.
SOURCE: Fluoride pollution of ground water, Discussion paper New Delhi, India, 1996.
ABSTRACT - National Library of Medicine
J Gastroenterol. 1996 Jun;31(3):333-7.
Gastroduodenal manifestations in patients with skeletal fluorosis.
Dasarathy S, Das TK, Gupta IP, Susheela AK, Tandon RK.
Department of Gastroenterology, All India Institute of Medical Sciences, Ansari Nagar, New Delhi, India.
A prospective case-controlled study was performed to evaluate the gastrointestinal symptoms and mucosal abnormalities occurring in patients with osteofluorosis. Ten patients with documented osteofluorosis and ten age- and sex-matched healthy volunteers were included in the study. Clinical evaluation, real-time ultrasound, and upper gastrointestinal endoscopy and biopsy from the gastric antrum and duodenum were performed in all subjects. The biopsies were subjected to a rapid urease test and light and electron microscopic examinations. Ionic fluoride levels were estimated in the drinking water, serum, and urine using an ION 85 ion analyzer. All patients with osteofluorosis had gastrointestinal symptoms, the most common being abdominal pain. Endoscopic abnormalities were found in seven patients with osteofluorosis. In all 7 of these patients, chronic atrophic gastritis was seen on histology. Electron microscopic abnormalities were observed in all 10 patients with osteofluorosis. These included loss of microvilli, cracked-clay appearance, and the presence of surface abrasions on the mucosal cells. None of the control subjects had any clinical symptoms or mucosal abnormalities. It was concluded that gastrointestinal symptoms as well as mucosal abnormalities are common in patients with osteofluorosis.
ABSTRACT - National Library of Medicine
J Clin Gastroenterol. 1994 Apr;18(3):194-9.
Toxic effects of chronic fluoride ingestion on the upper gastrointestinal tract.
Das TK, Susheela AK, Gupta IP, Dasarathy S, Tandon RK.
Department of Anatomy, All India Institute of Medical Sciences, New Delhi.
In a prospective case controlled study, we evaluated the adverse effects of long-term fluoride ingestion on the gastrointestinal tract. Ten patients with otosclerosis who were receiving sodium fluoride 30 mg/day for a period of 3-12 months, and 10 age- and sex-matched healthy volunteers were included. They were all evaluated clinically and subjected to a real time ultrasound examination, upper gastrointestinal endoscopy, and biopsies from the gastric antrum and duodenum. The biopsies were subjected to a rapid urease test as well as light and electron microscopic examinations. Ionic fluoride was estimated in the serum, urine, and drinking water using an ION 85 Ion Analyzer. Seven subjects (70%) ingesting fluoride had abdominal pain, vomiting, and nausea. Petechiae, erosions, and erythema were seen on endoscopy in all the subjects, but not in the controls. Histological examination of the gastric antral biopsy showed chronic atrophic gastritis in all the subjects but in only one (10%) healthy volunteer. Scanning electron microscopic examination showed "cracked-clay" appearance, scanty microvilli, surface abrasions, and desquamated epithelium in the subjects ingesting fluoride, but not in the controls. We conclude that long-term fluoride ingestion is associated with a high incidence of dyspeptic symptoms as well as histological and electron microscopic abnormalities.
ABSTRACT - National Library of Medicine
J Gastroenterol Hepatol. 1992 Jul-Aug;7(4):355-9.
Fluoride as a possible aetiological factor in non-ulcer dyspepsia.
Gupta IP, Das TK, Susheela AK, Dasarathy S, Tandon RK.
Department of Gastroenterology, All India Institute of Medical Sciences, New Delhi.
A prospective case controlled study was conducted to evaluate the role of fluoride as a possible aetiological factor for non-ulcer dyspepsia (NUD). Twenty patients with NUD and 10 age and sex matched healthy controls were subjected to clinical evaluation, upper gastrointestinal endoscopy and biopsies from the gastric antrum and duodenum. The antral and duodenal mucosa was subjected to a rapid urease test for Helicobacter pylori and histological and electron microscopic examinations. Fluoride levels in the drinking water, serum and urine were estimated using a ION 85 ion-analyser. These levels were significantly higher in patients with NUD than in controls (P less than 0.05). Histological abnormalities in the antral and duodenal mucosa were seen in 14 patients (70%) with NUD and 1 control subject (10%) (P less than 0.05). Electron microscopic abnormalities in the mucosal cells were seen in all patients with NUD but in none of the controls (P less than 0.01). The fluoride levels in serum and urine correlated with the symptoms, histological and electron microscopic abnormalities (P less than 0.05). It was concluded that chronic exposure to fluoride may result in NUD and should be considered in patients where other known cause of dyspepsia have been excluded.
ABSTRACT - National Library of Medicine
Prog Food Nutr Sci. 1986;10(3-4):279-314.
Skeletal fluorosis in humans: a review of recent progress in the understanding of the disease.
Krishnamachari KA.
Endemic skeletal fluorosis is a chronic metabolic bone and joint disease caused by ingesting large amounts of fluoride either through water or rarely from foods of endemic areas. Fluoride is a cumulative toxin which can alter accretion and resorption of bone tissue. It also affects the homeostasis of bone mineral metabolism. The total quantity of ingested fluoride is the single most important factor which determines the clinical course of the disease which is characterized by immobilization of joints of the axial skeleton and of the major joints of the extremities. A combination of osteosclerosis, osteomalacia and osteoporosis of varying degrees as well as exostosis formation characterizes the bone lesions. In a proportion of cases secondary hyperparathyroidism is observed with associated characteristic bone changes. Contrary to earlier thinking, severe crippling forms of skeletal fluorosis are seen in paediatric age group too. Increased metabolic turnover of the bone, impaired bone collagen synthesis and increased avidity for calcium are features in fluoride toxicity. Osteosclerotic picture is evident when small doses of fluoride are ingested over a long period of time during which calcium intakes are apparently normal while osteoporotic forms are common in paediatric age group and with higher body load of the element. Alterations in hormones concerned with bone mineral metabolism are seen in fluorosis. Kidney is the primary organ of excretion for fluorides. Age, sex, calcium intake in the diet, dose and duration of fluoride intake and renal efficiency in fluoride handling are the factors which influence the outcome. Serum parameters rarely help in the diagnosis. Elevated urinary fluoride and increased bone fluoride content are indicators of fluoride toxicity. Fluorosis is a preventable crippling disease. No effective therapeutic agent is available which can cure fluorosis. Industrial fluorosis is on the increase on a global basis. Bone density measurement is a tool for early diagnosis.